Since the atrioventricular (AV) node doesn't prevent all of these chaotic signals from entering the ventricles, the heart beats faster and irregularly.Ītrial fibrillation (A-fib) is an irregular and often very rapid heart rhythm (arrhythmia) that can lead to blood clots in the heart. In atrial fibrillation, electrical signals fire from multiple locations in the atria (typically pulmonary veins), causing them to beat chaotically. The signal then travels through the atria to the atrioventricular (AV) node and passes into the ventricles, causing them to contract and pump out blood. El paciente fué visto la última vez con la edad de un año y tres semanas.In a typical heart, a tiny cluster of cells at the sinus node sends out an electrical signal. La frecuencia ventricular permaneció aproximadamente en 200 y había una respuesta ventricular de 2:1. El tratamiento con digital, quinidina, procainamida y prostigmina no dieron resultado. No había señales positivos de malformación asociada del corazón. La historia del caso, y la persistencia del transtorno están fuertemente a favor de au origen congénito. El transtorno del ritmo fué diagnosticado por medio del electrocardiograma cuando el paciente tenía cuatro meses y medio de edad. Se refiere el caso de un aleteo auricular persistente en un lactante. Der Patient wurde zuletzt im Alter von 1 Jahr und 3 Monaten gesehen. Die Kammerfrequenz blieb annähemd 200 bei einein 2:1 Block. Die Behandlung mit Digitalis, Chinidin, Procamid und Prostiginin hatte keinen Effekt. Es gab kein positives Zeichen für gleichzeitige Missbildungen des Herzens. Die Krankengeschichte und das Persistieren des Flatterns sprechen streng für eine kongenitale Genese. Die Arythmie wurde im Alter von 4½ Monaten bei dem Kinde mit Hilfe des EKG diagnostiziert. Le patient a été vu une dernière fois à l'äge d'un an et trois semaines.Įin Fall eines andauernden Vorhofflatterns bei einem Kind wird besprochen. Le rythme ventriculaire restait aux environs de 200 avec une réponse du ventricule de 2:1. Un traitement à la digitaline, la quinidine, la procamide et la prostigmine n'avait aucun effet. On n'a pas trouvé de signes certains d'une malformation cardiaque associée. L'histoire de la maladie et la persistance du flutter étaient des arguments importants en faveur d'une origine congénitale. Le diagnostic de l'arythmie a été poséà l'âge de quatre mois et demie grâce à l'électro-cardiogramme. L'auteur rapporte un cas de flutter auriculaire persistant chez un nourrisson. The patient was last seen at the age of 1 year and 3 weeks. The ventricular rate remained approximately at 200 and there was a 2:1 ventricular response. A total of 34 electrocardiographic examinations, supplemented by phonocardiographic studies, were performed during a period of observation of 9 months. Treatment with digitalis, quinidine, Procamide and Prostigmin had no effect. There was no positive evidence of associated malformations of the heart. The case history and the persistent nature of the flutter strongly favoured a congenital origin. The arrhythmia was diagnosed by means of electrocardiography when the patient was 4½ months of age. SummaryĪ case of persistent auricular flutter in an infant is reported. The case reported herein not only adds to the small number of cases previously described but presents some unusual features, inasmuch as the flutter persisted over a period of observation of nine months. In nine of these cases, a normal rhythm was restored. A review of the literature has revealed solitary additional cases (M annheimer 1940, H edberg 1945, L andtman 1947, and M cL ean 1952). To this number they added one case of their own. In 1960, L each and G ibson found reports of only six cases of this arrhythmia in infants under one year of age.
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